Nace Golding

Golding, Nace
Associate Professor in Neurobiology

E-mail: golding@mail.utexas.edu

Website: http://www.utexas.edu/neuroscience/Neurobiology/NaceGolding/index.html

Main Office: PAT 407
Phone: 232-4888

Alternate Office: PAT 408
Phone: 232-4889

Mailing Address:
The University of Texas at Austin
1 University Station C0920
Austin, TX 78712-1095

Graduate Students:

  • Khurana, Sukant

    • Research Summary:
       Neuronal dendrites are elaborate, tree-like structures that receive up to thousands of excitatory and inhibitory synaptic connections. The morphology and electrical properties of the dendrites strongly influence the way in which synaptic activity sums together and is ultimately translated into new patterns of action potential firing in the axon. This translation process, called dendritic integration, is the fundamental means by which neurons regulate what synaptic information is communicated to the neuron's network targets. My research focuses on identifying the mechanisms by which dendrites shape synaptic activity and action potential firing, as well as understanding how these mechanisms contribute to the neuron's functional role. An additional focus is on how dendritic properties influence changes in synaptic strength that, in turn, underlie some forms of learning and memory.
     
    Publications:
    Weak action potential backpropagation is associated with high frequency axonal firing capability in principal neurons of the gerbil medial superior olive (2007) Journal of Physiology 583, 647-661.
    Factors mediating powerful voltage attenuation along CA1 dendrites. (2005) Journal of Physiology 568, 69-82.
    Post-hearing developmental refinement of temporal processing in principal neurons of the medial superior olive. (2005) Journal of Neuroscience 25, 7887-7895.
    Dendritic spikes as a mechanismfor cooperative long-term potentiation (2002) Nature 418, 326-331.
    Dichotomy of action potential backpropagation in CA1 pyramidal neuron dendrites (2001) Journal of Neurophysiology 86, 2998-3010.

     
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